Brain Toniq Review: The Science Behind the Think Drink

The ability to multitask and mentally juggle multiple demands is essential in today’s fast-paced world. At the same time, we’re bombarded with information that can both distract and overload our focus and attention.

Many of us need a caffeine “boost” in the morning or throughout the day to maintain mental focus. However, drinking too much coffee or tea leaves you feeling like you need to do a couple of laps around the building.

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And although coffee consumption offers a number of potential health benefits, many of us drink more than enough of it on a daily basis. Energy drinks are an alternative option. However, their effects on cognitive performance are principally related to the presence of caffeine [1].

Enter Brain Toniq

Brain Toniq bills itself as the world’s first and only botanical-based, non-caffeinated functional “think drink”, specifically designed to increase mental focus, function and clarity. According to the Brain Toniq website:

Formulated out of plant extracts and natural compounds, the ingredients in Brain Toniq have a long, proven history for their effects on increasing brain power and cognition.

I’d previously heard about Brain Toniq and was intrigued at the idea of an energy drink designed to increase cognitive performance. Additionally, the Brain Toniq website references peer-reviewed research studies that examine many of the ingredients. When I contacted the company, they were kind enough to send me a sample to review.

Neurodegenerative Disease and the Coming Epidemic

At Neuroscience 2008, the 38th annual meeting of the Society for Neuroscience held last month in Washington D.C., a number of researchers presented evidence that a small, soluble, clustered form of a protein called amyloid beta may be responsible for brain damage in Alzheimer’s disease patients [1]. In addition, scientists report that they are finding new sources and uses of neural stem cells that may replace cells damaged by neurodegenerative disorders such as Alzheimer’s and Parkinson’s disease [2].

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Creative Commons License photo credit: neurollero

Why are these reports significant? Because until recently, large insoluble amyloid beta plaques, or deposits, were considered the likely cause of Alzheimer’s disease. The plaques were thought to disrupt brain cell communication. However, new findings show that an early (i.e. small), soluble, clustered form of amyloid beta called protofibrils is found in high levels in the brains of people with Alzheimer’s disease [1]. Researchers also found a strong correlation between the presence of high levels of protofibrils in the brains of transgenic mouse models of Alzheimer’s disease and the cognitive impairments associated with the disease.